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Thursday, 28 November 2013

Glaucoma Preventions – The do and do not’s list

Glaucoma is a medical condition of an eye disease as result of the damage of the nerve of eye’s optic over time. If left untreated, it can lead to permanently vision impair and blindness.
V. Preventions
A. The do and do not’s list
1. Eye yearly examination
The exam can detect any eye disease in early stage. It is recommended that people to have regular eye examination every year and comprehensive eye examination every three to five years.
2. Avoid Nutrient deficiency
There is a report of Rats fed a Vit E-deficient diet with surgically induced intraocular pressure (IOP) elevation experience significantly more retinal ganglion cells (RGCs) death than rats fed a normal diet. This phenomenon may be related to the increased level of lipid peroxidation in Vit E-deficient rats(36). Vitamin A, B(9), C, E, and uric acid are well-known antioxidants and may prevent age-related eye disorders, according to the study by, normal-tension glaucoma patients had lower serum levels of vitamin C and increased levels of uric acid. These observations may pave the way for possible alternative treatment for normal-tension glaucoma(37).
3. Avoid vitamin A deficiency
In the study to investigate the prevalence and causes of blindness in sub-Saharan Africa, the existing services and limitations, and the Vision 2020 goals for the future, showed that the bulk of blindness in the region is preventable or curable. Efforts should focus on eye problems which are universally present and for which there are cost effective remedies, such as cataract and refractive problems and on those problems which occur focally and can be prevented by primary healthcare measures, such as trachoma, onchocerciasis, and vitamin A deficiency(38).
4. Glaucoma eyedrops
If you are experience Elevated eye pressurem, some researchers suggested the use of Glaucoma eyedrops as it can significantly reduce the risk that elevated eye pressure of that will progress to glaucoma.
5. Prevention eye damage
There are some reports that serious eye injuries can lead to glaucoma, such as a problem secondary posttraumatic glaucoma caused by perforative eye injuries or contusions of the eyeball(39). or Secondary glaucoma in eye injuries complicated by trauma of the crystalline lens(40).
6. Lower your iblood sugar levels
If you are diabetic, low your sugar levels may reduce the risk of Glaucoma, as diabetes mellitus contributes to an earlier and a quicker development of opacities of the lens and worsening of the vision in affected people(41).
7. Exercise regularly or not
In diabetic rat study, moderate physical exercise has marked anti-inflammatory effects. This may be an efficient strategy to protect diabetics against microorganism infection, insulin resistance and vascular complications(42), but according to the study by the University of Iowa Hospitals and Clinics, although the authors do not believe that all patients with the pigmentary dispersion syndrome or pigmentary glaucoma need to avoid exercise, however, for patients with these disorders who regularly engage in jogging or more strenuous or more jarring types of exercise, they suggest an evaluation before and after the type of exercise in question. If marked exercise-induced pigment dispersion occurs, pilocarpine therapy may be an alternative to avoidance of the exercise(43).
8. Lifestyle
According to the review by the Massachusetts Eye and Ear Infirmar, various modifiable lifestyle factors, such as exercise, diet, and cigarette smoking, that may influence intraocular pressure and that have been studied in relation to the risk of developing POAG(43a).
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Sources
(a) tp://www.glaucoma.org/glaucoma/types-of-glaucoma.php
(36) http://www.ncbi.nlm.nih.gov/pubmed/20795867
(37) http://www.ncbi.nlm.nih.gov/pubmed/19763599
(38) http://www.ncbi.nlm.nih.gov/pubmed/11466240
(39) http://www.ncbi.nlm.nih.gov/pubmed/7934236
(40) http://www.ncbi.nlm.nih.gov/pubmed/3627661
(41) http://www.ncbi.nlm.nih.gov/pubmed/21121128
(42) http://www.ncbi.nlm.nih.gov/pubmed/20846161
(43) http://www.ncbi.nlm.nih.gov/pubmed/1495789
(43a) http://www.ncbi.nlm.nih.gov/pubmed/19680048